Bruce Teter, Ph.D.

Titles

Associate Professor, Geriatric Medicine
Member, Brain Research Institute
Adjunct Associate Professor, Department of Medicine

Contact Information

Email
bteter@ucla.edu
Phone
Cell-phone Number: 3104131759Fax Number: 8188955835Work Phone Number: (818) 891-7711 x7481 8188917711x7645
Address
Mailing Address: VA Greater Los Angeles Healthcare System, SepulvedaDepartment of Medicine16111 Plummer StreetMC 151Sepulveda, CA 91343UNITED STATES Work Address: OfficeUCLA-VAMCmc15116111 Plummer St.Sepulveda, CA 91343UNITED STATES LaboratorySEPUL VAMC 7Los Angeles, CA 90095UNITED STATES

Biography

Bruce Teter received a B.S. in marine biology and a minor in chemistry from Cal. State U., Long Beach in 1982. Dr. Teter received a Ph.D. degree in molecular biology from the University of Southern California, Los Angeles, in 1991. He then completed a postdoctoral fellowship in neurogerontology working with Dr. Caleb Finch at the Andrus Gerontology Center in the School of Gerontology at USC. In 1992, Dr. Teter was appointed Research Associate at the Andrus Gerontology Center. In 1996 he joined the lab of Dr. Greg Cole at UCLA and is currently Associate Professor in the Department of Medicine at UCLA, and Chief of the ApoE Research Laboratory, and Research Associate at GRECC, Veterans Administration, GLAHS.

Publications:

Teter, B.   Life-span influences of apoE4 on CNS function. Invited Peer Commentary on: Schonheit, B., Glockner, F., and Ohm, T.G. (2006) Apolipoprotein E polymorphism and dendritic shape. , Neurobiology of Aging, 2006; in press: .
Teter, B., Finch, C.E.   Caliban's Inheritance: Genetics of Neuronal Aging, Trends in Neuroscience , 2004; 10: 627-32.
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Teter B, Xu P-T, Gilbert JR, Roses AD, Galasko D, Cole GM   Defective neuronal sprouting supported by human Apolipoprotein E4 represents a gain-of-deleterious function, J. Neurosci. Res, 2002; (687): 331-336.
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Teter B, Ashford JW   Neuroplasticity in Alzheimer's Disease, Journal of Neuroscience Research, 2002; 70(Aging Brain and Alzheimer's Disease Special Issue): 402-437.
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Teter B, Raber J, Nathan B, Crutcher KA   The presence of apoE4, not the absence of apoE3, contributes to AD pathology, J. Alzheimer's Disease, 2002; (4): 155-163.